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Figure 22-52 A Schiller-Duval body in yolk sac carcinoma.

Figure 22-53Granulosa cell tumor. A, The tumor cells are arranged in sheets punctuated by small follicle-like structures (Call-Exner bodies). B, Strong immunohistochemical positivity

with an antibody to inhibin characterizes these tumors.

Figure 22-54 A, Thecoma-fibroma composed of plump, differentiated stromal cells with thecal appearance. B, Large bisected fibroma of the ovary apparent as a white, firm mass (right).

The fallopian tube is attached.

Figure 22-55Sertoli cell tumor. A, Gross photograph illustrating characteristic golden yellow appearance of the tumor. B, Photomicrograph showing well-differentiated Sertoli cell tubules.

(Courtesy of Dr. William Welch, Brigham and Women's Hospital, Boston, MA.)

Figure 22-56Potential sites for ectopic pregnancy, including the fallopian tube, ovary, cornu, and (rarely) abdominal viscera.

Figure 22-57 A, Diagram of placental anatomy. Within the outer boundary of myometrium is a layer of decidua, from which the maternal vessels originate and deliver blood to and from

the intervillous spaces. Umbilical vessels branch and terminate in placental villi, where nutrient exchange takes place. B, Normal term placenta (fetal surface) with umbilical cord.

 

Figure 22-58Diagrammatic representation of the various types of twin placentation and their membrane relationships. (Adapted from Gersell D, et al: Diseases of the placenta. In

Kurman, R (ed): Blaustein's Pathology of the Female Genital Tract. New York, Springer-Verlag, 1994.)

Figure 22-59Twin-twin transfusion syndrome resulting in the death of both fetuses because of excessive (left) or deficient (right) blood volume.

Figure 22-60Placental infections derived from ascending and blood-borne routes. Acute chorioamnionitis. A, On gross examination, the placenta contains greenish opaque membranes.

Compare with Figure 22-55B . B, A photomicrograph illustrates a dense band-like inflammatory exudate on the amniotic surface (top). C, Acute necrotizing intervillositis, from a fetalmaternal

infection by listeria.

Figure 22-61Proposed sequence of events in the pathogenesis of toxemia of pregnancy. The main features are (1) decreased uteroplacental perfusion; (2) increased vasoconstrictors and

decreased vasodilators, resulting in local and systemic vasoconstriction; and (3) disseminated intravascular coagulation (DIC). (Adapted from Friedman SA: Pre-eclampsia: a review of the

role of prostaglandins. Obstet Gynecol 71:122, 1988. Reprinted by permission of the American College of Obstetricians and Gynecologists; and Khong TY, et al: Inadequate maternal

vascular response to placentation in pregnancies complicated by pre-eclampsia and by small for gestational age infants. Br J Obstet Gynecol 93:1049, 1986.)

Figure 22-62Acute atherosis of uterine vessels in eclampsia. Note fibrinoid necrosis of the vessel walls, subendothelial macrophages and perivascular lymphocytic infiltrate. (Courtesy of

Dr. Drucilla J. Roberts, Massachusetts General Hospital, Boston, MA.)



TABLE 22-5-- Features of Complete Versus Partial Hydatidiform Mole


Date: 2016-04-22; view: 761


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