Tasks for self-control

Task № 1. At the section about 2 liters of purulent fluid were revealed in the abdominal cavity. The peritoneum is not transparent, there is gray fibrin mass on the peritoneum surface, these fibrin mass covers also the loops.

a) What pathological process is it?

b) Describe the exudate composition.

c) Describe cells, characteristic for it.

Task № 2. By examining with the laryngoscope of the removed tonsils hyperemia, swelling, ulcers and fibrinous film on the mucous tunic surface were revealed.

a) What pathological process is it?

b) Detect the type of exudative inflammation.

c) Describe the exudate composition.

Task № 3. A patient with chronic glomerulonephritis, 38 years, died from uremia. At the section the gray-yellow “hairy” pericardial surface was revealed. There was a gray film on visceral pleura that could be easy removed.

a) Detect the type of exudative inflammation.

b) Name the inflammation types occurung on the pericardial and visceral pleura.

c) Describe the epicardial changes in this pathology.

Task № 4. A patient, 47 years, died from focal hypostatic bronchopneumonia (intraarticular cervical hip fracture). At the section 2 liters of muddy yellow fluid were revealed in the right pleural cavity. Pleura was swollen and friable.

a) Detect the type of exudative inflammation.

b) Name varieties of this inflammation;

c) Detect the consequences of this process.

Task № 5. A patient, 63 years, died from fibrocavernous tuberculosis. At the section the cavity 50mm in diameter with a tight wall was revealed in the lungs. Inside of the cavity there was greenish-yellow dry caseous mass. The kidneys were gray, enlarged and indurated. The cortical layer was wide, pyramids were flat and half-transparant.

a) What pathological process is it?

b) Detect the character of morphological changes.

c) Name the pathology of the kidneys.

The answers.

Task № 1.

a)purulent fibrinous peritonitis;

b) polymorphonuclear leukocytes, fibrin, mesotheliocytes;

c) purulent bodies — leucocytes in fatty degeneration.

Task № 2.

a) angina;

b) fibrinous inflammation (diphtheritic);

c) fibrin, polymorphonuclear leukocytes.

Task № 3.

a) fibrinous inflammation;

b) croupous fibrinous inflammation;

c) Cor villosum or “hairy heart”.

Task № 4.

a) purulent pleuritis;

b) abscess, phlegmon;

c) resorption, chronic form (pleural empyema).

Task № 5.

a) chronic lung cavity;

b) kidneys amyloidosis;

c) stromal proteinosis.

Lesson № 9

Methodical instruction for students

Theme: PRODUCTIVE INFLAMMATION. IMMUNOPATHOLOGIC PROCESSES.

Aim: to study etiology, pathogenesis, morphology and consequences for the organism of the productive inflammation.

Objectives of the lesson:

1. To study the definition and the classification of productive inflammation.

2. To study etiology and pathogenesis of productive inflammation.

3. To study the consequences and significance for organism of productive inflammation.

4. To study the definition and classification of immunopathologic processes.

5. To study morphology characteristic of immediate-type hypersensitivity and delayed-type hypersensitivity reactions.

6. To study the classification of immunodeficiency syndromes.

Date: 2016-03-03; view: 826